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Sentence processing in aphasia: Evi...

Nakano, Hiroko.

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Sentence processing in aphasia: Evidence for deficits in incremental combinatorial thematics.

Record Type:	Language materials, printed : Monograph/item
Title/Author:	Sentence processing in aphasia: Evidence for deficits in incremental combinatorial thematics./
Author:	Nakano, Hiroko.
Description:	194 p.
Notes:	Adviser: Sheila E. Blumstein.
Contained By:	Dissertation Abstracts International62-02B.
Subject:	Language, Linguistics. -
Online resource:	http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3006771
ISBN:	9780493160825

Sentence processing in aphasia: Evidence for deficits in incremental combinatorial thematics.
Nakano, Hiroko.

Sentence processing in aphasia: Evidence for deficits in incremental combinatorial thematics. - 194 p.

Adviser: Sheila E. Blumstein.

Thesis (Ph.D.)--Brown University, 2001.

The process of combinatorial thematics in aphasia was investigated to better characterize both Broca's and Wernicke's aphasics' sentence comprehension deficits. The primary question was how aphasics integrate lexical-semantic and pragmatic information with a canonical simple active syntactic structure to form an appropriate thematic interpretation of the sentence. In Study I (off-line incremental thematic judgment experiments), in which subjects' explicit thematic knowledge was elicited, both Broca's and Wernicke's aphasics showed evidence of general incremental combinatorial thematics, integrating syntactic, verb-semantic, and pragmatic information, thereby demonstrating overall correct judgements between plausible and implausible events. However, both Broca's and Wernicke's aphasics failed to show sensitivity to fine-grained thematic structures, displaying no appreciation of subtle differences in the degree of plausibility in plausible events. Study II (on-line sentential thematic priming experiments), which elicited subjects' implicit thematic knowledge, revealed distinctly impaired combinatorial processing in the Broca's and Wernicke's aphasic groups. Results showed that Broca's aphasics had 'reduced sensitivity', whereas Wernicke's aphasics had 'overly activated' processing in combinatorial thematics. Both groups showed no evidence of integrating thematic information derived from a subject noun phrase and verb cluster to generate a relevant thematic structure in a simple active sentence. Based on the off-line and on-line data, it was concluded that both Broca's and Wernicke's aphasics have deficits, with distinct deficit patterns, in integrating thematic information from lexical-semantics and pragmatics in a given syntactic structure. The Broca's aphasics' deficit was considered to be attributable to a reduced activation of the lexicon, a slowed activation of the lexicon and/or syntactic operations, or a reduced working memory capacity, while the Wernicke's aphasics' deficit was hypothesized to be due to an over-activation of the lexicon or disrupted inter-nodal connections. Finally, these results point to the necessity for the investigation of the cognitive/neural mechanisms that underlie the process of combinatorial thematics in order to reach a more conclusive understanding of the mechanisms for sentence comprehension deficits in aphasia.

ISBN: 9780493160825Subjects--Topical Terms:

1018079
Language, Linguistics.

Sentence processing in aphasia: Evidence for deficits in incremental combinatorial thematics.
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100 1 $a Nakano, Hiroko. $3 1294388
245 1 0 $a Sentence processing in aphasia: Evidence for deficits in incremental combinatorial thematics.
300 $a 194 p.
500 $a Adviser: Sheila E. Blumstein.
500 $a Source: Dissertation Abstracts International, Volume: 62-02, Section: B, page: 1110.
502 $a Thesis (Ph.D.)--Brown University, 2001.
520 $a The process of combinatorial thematics in aphasia was investigated to better characterize both Broca's and Wernicke's aphasics' sentence comprehension deficits. The primary question was how aphasics integrate lexical-semantic and pragmatic information with a canonical simple active syntactic structure to form an appropriate thematic interpretation of the sentence. In Study I (off-line incremental thematic judgment experiments), in which subjects' explicit thematic knowledge was elicited, both Broca's and Wernicke's aphasics showed evidence of general incremental combinatorial thematics, integrating syntactic, verb-semantic, and pragmatic information, thereby demonstrating overall correct judgements between plausible and implausible events. However, both Broca's and Wernicke's aphasics failed to show sensitivity to fine-grained thematic structures, displaying no appreciation of subtle differences in the degree of plausibility in plausible events. Study II (on-line sentential thematic priming experiments), which elicited subjects' implicit thematic knowledge, revealed distinctly impaired combinatorial processing in the Broca's and Wernicke's aphasic groups. Results showed that Broca's aphasics had 'reduced sensitivity', whereas Wernicke's aphasics had 'overly activated' processing in combinatorial thematics. Both groups showed no evidence of integrating thematic information derived from a subject noun phrase and verb cluster to generate a relevant thematic structure in a simple active sentence. Based on the off-line and on-line data, it was concluded that both Broca's and Wernicke's aphasics have deficits, with distinct deficit patterns, in integrating thematic information from lexical-semantics and pragmatics in a given syntactic structure. The Broca's aphasics' deficit was considered to be attributable to a reduced activation of the lexicon, a slowed activation of the lexicon and/or syntactic operations, or a reduced working memory capacity, while the Wernicke's aphasics' deficit was hypothesized to be due to an over-activation of the lexicon or disrupted inter-nodal connections. Finally, these results point to the necessity for the investigation of the cognitive/neural mechanisms that underlie the process of combinatorial thematics in order to reach a more conclusive understanding of the mechanisms for sentence comprehension deficits in aphasia.
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