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Cercospora Leaf Spot in Sugar Beet. ...

Vereijssen, Jessica,

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Cercospora Leaf Spot in Sugar Beet. Epidemiology, Life Cycle Components and Disease Management /

Record Type:	Electronic resources : Monograph/item
Title/Author:	Cercospora Leaf Spot in Sugar Beet. Epidemiology, Life Cycle Components and Disease Management // Jessica Vereijssen.
Author:	Vereijssen, Jessica,
Description:	1 electronic resource (212 pages)
Notes:	Source: Dissertations Abstracts International, Volume: 83-02, Section: B.
Contained By:	Dissertations Abstracts International83-02B.
Subject:	Agriculture. -
Online resource:	https://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=28239079
ISBN:	9798516028533

Cercospora Leaf Spot in Sugar Beet. Epidemiology, Life Cycle Components and Disease Management /
Vereijssen, Jessica,

Cercospora Leaf Spot in Sugar Beet. Epidemiology, Life Cycle Components and Disease Management /Jessica Vereijssen. - 1 electronic resource (212 pages)

Source: Dissertations Abstracts International, Volume: 83-02, Section: B.

Cercospora beticola, the causal agent of Cercospora leaf spot (CLS) in sugar beet, causes reductions in sugar yield and financial returns. Section I describes the epidemiology of CLS. Two disease severity indices were compared and their relation with sugar yield modelled. A linear curve fitted the Agronomica disease severity index with sugar yield in both severely and mildly infected fields, whereas for a leaf disease severity assessment this relationship was exponential and linear, respectively. The Agronomica disease severity index was less laborious and improved disease assessment. Using spatial and temporal models, the within-row dependencies of CLS disease severities was demonstrated. However, disease increase on a given plant was largely determined by disease on that plant. Similarly when describing the within-plant dynamics of disease, it was concluded that the major determinant of disease increase on a given leaf layer on a plant was from disease on that leaf layer. Spraying a fungicide twice changed the contribution of the lower leaf layers. In one mildly infected field, these leaf layers were more often significant in the model; in the other severely infected field less often. In section II studies on the life cycle of CLS are reported. It was demonstrated that C. beticola is able to infect sugar beet through the root in glasshouse, climate room, and in field experiments. The genetic variability of C. beticola was studied in an international collection of isolates. Between and within geographic regions, genetic variability is high. A C. beticola specific PCR primer was developed, which enabled detection of C. beticola in plant tissue. We showed systemic growth of C. beticola in a sugar beet seedling after root infection. Section III describes the development of a supervised control system. The use of an action threshold involving two early sprays and weather-based treatments resulted in sugar yields and financial returns similar to calendar sprayed treatments, and saved up to two fungicide applications.

English

ISBN: 9798516028533Subjects--Topical Terms:

518588
Agriculture.

Cercospora Leaf Spot in Sugar Beet. Epidemiology, Life Cycle Components and Disease Management /
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520 $a Cercospora beticola, the causal agent of Cercospora leaf spot (CLS) in sugar beet, causes reductions in sugar yield and financial returns. Section I describes the epidemiology of CLS. Two disease severity indices were compared and their relation with sugar yield modelled. A linear curve fitted the Agronomica disease severity index with sugar yield in both severely and mildly infected fields, whereas for a leaf disease severity assessment this relationship was exponential and linear, respectively. The Agronomica disease severity index was less laborious and improved disease assessment. Using spatial and temporal models, the within-row dependencies of CLS disease severities was demonstrated. However, disease increase on a given plant was largely determined by disease on that plant. Similarly when describing the within-plant dynamics of disease, it was concluded that the major determinant of disease increase on a given leaf layer on a plant was from disease on that leaf layer. Spraying a fungicide twice changed the contribution of the lower leaf layers. In one mildly infected field, these leaf layers were more often significant in the model; in the other severely infected field less often. In section II studies on the life cycle of CLS are reported. It was demonstrated that C. beticola is able to infect sugar beet through the root in glasshouse, climate room, and in field experiments. The genetic variability of C. beticola was studied in an international collection of isolates. Between and within geographic regions, genetic variability is high. A C. beticola specific PCR primer was developed, which enabled detection of C. beticola in plant tissue. We showed systemic growth of C. beticola in a sugar beet seedling after root infection. Section III describes the development of a supervised control system. The use of an action threshold involving two early sprays and weather-based treatments resulted in sugar yields and financial returns similar to calendar sprayed treatments, and saved up to two fungicide applications.
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720 1 $a Jeger, M. J. $e degree supervisor.
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