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Inflammation and epilepsy = new vistas /

Janigro, Damir.

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Inflammation and epilepsy = new vistas /

Record Type:	Electronic resources : Monograph/item
Title/Author:	Inflammation and epilepsy/ edited by Damir Janigro, Astrid Nehlig, Nicola Marchi.
Reminder of title:	new vistas /
other author:	Janigro, Damir.
Published:	Cham :Springer International Publishing : : 2021.,
Description:	vii, 237 p. :ill., digital ;24 cm.
[NT 15003449]:	Pro and Anti-Inflammatory Neurovascular Processes in Epilepsy: A Fragile and Dynamic Equilibrium -- Emerging Molecular Mechanisms of Neuroinflammation in Seizure Disorders -- Glial Mechanisms of Inflammation during Seizures -- Perivascular Inflammation and Extracellular Matrix Alterations in Blood-Brain Barrier Dysfunction and Epilepsy -- Blood T cells and Cytokine Levels during Interictal-Ictal Transitions -- Autoantibodies, Encephalopathies and Epilepsy -- Tau Protein in Drug-Resistant Epilepsy and Cognitive Decline -- Ketogenic Diet, Inflammation and Epilepsy -- Role of Regulatory T cells in Epilepsy -- Inflammation at the Neurovascular Unit in Post-Traumatic Epilepsy.
Contained By:	Springer Nature eBook
Subject:	Epilepsy. -
Online resource:	https://doi.org/10.1007/978-3-030-67403-8
ISBN:	9783030674038

Inflammation and epilepsy = new vistas /
Inflammation and epilepsynew vistas /[electronic resource] :edited by Damir Janigro, Astrid Nehlig, Nicola Marchi. - Cham :Springer International Publishing :2021. - vii, 237 p. :ill., digital ;24 cm. - Progress in inflammation research,v.880379-0363 ;. - Progress in inflammation research ;v.88..

Pro and Anti-Inflammatory Neurovascular Processes in Epilepsy: A Fragile and Dynamic Equilibrium -- Emerging Molecular Mechanisms of Neuroinflammation in Seizure Disorders -- Glial Mechanisms of Inflammation during Seizures -- Perivascular Inflammation and Extracellular Matrix Alterations in Blood-Brain Barrier Dysfunction and Epilepsy -- Blood T cells and Cytokine Levels during Interictal-Ictal Transitions -- Autoantibodies, Encephalopathies and Epilepsy -- Tau Protein in Drug-Resistant Epilepsy and Cognitive Decline -- Ketogenic Diet, Inflammation and Epilepsy -- Role of Regulatory T cells in Epilepsy -- Inflammation at the Neurovascular Unit in Post-Traumatic Epilepsy.

This book provides comprehensive information, both for clinicians and scientists, on the basic mechanisms, clinical features, and therapeutic approaches to epilepsy as an inflammatory disease. Inflammation has been for many years considered as an etiologic player (and a therapeutic target) for a specific group of epilepsies. However, it turns out that this concept underestimated the impact of inflammation in seizure disorders. Many accepted therapies for non-inflammatory epilepsies act in part as an inflammatory drug. The CNS actively responds to acute immune challenges by altering body temperature, stimulating the HPA axis, as well as up- and down-regulating specific sympathetic pathways.

ISBN: 9783030674038

Standard No.: 10.1007/978-3-030-67403-8doiSubjects--Topical Terms:

743095
Epilepsy.

LC Class. No.: RC372 / .I54 2021

Dewey Class. No.: 616.853

Inflammation and epilepsy = new vistas /
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490 1 $a Progress in inflammation research, $x 0379-0363 ; $v v.88
505 0 $a Pro and Anti-Inflammatory Neurovascular Processes in Epilepsy: A Fragile and Dynamic Equilibrium -- Emerging Molecular Mechanisms of Neuroinflammation in Seizure Disorders -- Glial Mechanisms of Inflammation during Seizures -- Perivascular Inflammation and Extracellular Matrix Alterations in Blood-Brain Barrier Dysfunction and Epilepsy -- Blood T cells and Cytokine Levels during Interictal-Ictal Transitions -- Autoantibodies, Encephalopathies and Epilepsy -- Tau Protein in Drug-Resistant Epilepsy and Cognitive Decline -- Ketogenic Diet, Inflammation and Epilepsy -- Role of Regulatory T cells in Epilepsy -- Inflammation at the Neurovascular Unit in Post-Traumatic Epilepsy.
520 $a This book provides comprehensive information, both for clinicians and scientists, on the basic mechanisms, clinical features, and therapeutic approaches to epilepsy as an inflammatory disease. Inflammation has been for many years considered as an etiologic player (and a therapeutic target) for a specific group of epilepsies. However, it turns out that this concept underestimated the impact of inflammation in seizure disorders. Many accepted therapies for non-inflammatory epilepsies act in part as an inflammatory drug. The CNS actively responds to acute immune challenges by altering body temperature, stimulating the HPA axis, as well as up- and down-regulating specific sympathetic pathways.
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650 2 4 $a Neurosciences. $3 588700
650 2 4 $a Infectious Diseases. $3 894100
650 2 4 $a Pharmacology/Toxicology. $3 890953
700 1 $a Janigro, Damir. $3 3495210
700 1 $a Nehlig, Astrid. $3 915473
700 1 $a Marchi, Nicola. $3 3495211
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