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Histophilus somni = biology, molecul...

Inzana, Thomas J.

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Histophilus somni = biology, molecular basis of pathogenesis, and host immunity /

Record Type:	Electronic resources : Monograph/item
Title/Author:	Histophilus somni/ edited by Thomas J. Inzana.
Reminder of title:	biology, molecular basis of pathogenesis, and host immunity /
other author:	Inzana, Thomas J.
Published:	Cham :Springer International Publishing : : 2016.,
Description:	vii, 160 p. :ill., digital ;24 cm.
[NT 15003449]:	Taxonomy of Histophilus somni -- Histophilosis as a Natural Disease -- Histophilus somni Genomics and Genetics -- Interactions on Histophilus somni with Host Cells -- Histophilus somni Surface Proteins -- Host Immune Response to Histophilus somni -- The Many Facets of Lipooligosaccharide as a Virulence Factor of Histophilus somni -- Exopolysaccharide Production and Biofilm Formation by Histophilus somni.
Contained By:	Springer eBooks
Subject:	Bacterial diseases in animals. -
Online resource:	http://dx.doi.org/10.1007/978-3-319-29556-5
ISBN:	9783319295565

Histophilus somni = biology, molecular basis of pathogenesis, and host immunity /
Histophilus somnibiology, molecular basis of pathogenesis, and host immunity /[electronic resource] :edited by Thomas J. Inzana. - Cham :Springer International Publishing :2016. - vii, 160 p. :ill., digital ;24 cm. - Current topics in microbiology and immunology,v.3960070-217X ;. - Current topics in microbiology and immunology ;v.351..

Taxonomy of Histophilus somni -- Histophilosis as a Natural Disease -- Histophilus somni Genomics and Genetics -- Interactions on Histophilus somni with Host Cells -- Histophilus somni Surface Proteins -- Host Immune Response to Histophilus somni -- The Many Facets of Lipooligosaccharide as a Virulence Factor of Histophilus somni -- Exopolysaccharide Production and Biofilm Formation by Histophilus somni.

This volume reviews the current understanding of the taxonomy, disease syndromes, genetics, biology, and pathogenic factors of Histophilus somni, as well as the host immune response to this pathogen. H. somni is one of the most important bacterial pathogens in cattle and other ruminants, and its virulence factors are highly conserved with Haemophilus influenzae and other members of the Pasteurellaceae. H. somni has been recognized as a major cause of thrombotic meningoencephalitis, respiratory disease syndromes, myocarditis, reproductive disease syndromes, polyarthritis, mastitis, ocular disease, and septicemia. The only known habitats of H. somni are the mucosal surfaces of ruminants, making this bacterium an opportunistic pathogen. Although it is capable of causing inflammation at systemic sites and is toxic to epithelial and phagocytic cells, the bacterium's wide array of virulence factors act primarily as a defense against, or to escape recognition from, host innate and adaptive immunity.

ISBN: 9783319295565

Standard No.: 10.1007/978-3-319-29556-5doiSubjects--Topical Terms:

2191118
Bacterial diseases in animals.

LC Class. No.: SF780.3

Dewey Class. No.: 571.99311

Histophilus somni = biology, molecular basis of pathogenesis, and host immunity /
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520 $a This volume reviews the current understanding of the taxonomy, disease syndromes, genetics, biology, and pathogenic factors of Histophilus somni, as well as the host immune response to this pathogen. H. somni is one of the most important bacterial pathogens in cattle and other ruminants, and its virulence factors are highly conserved with Haemophilus influenzae and other members of the Pasteurellaceae. H. somni has been recognized as a major cause of thrombotic meningoencephalitis, respiratory disease syndromes, myocarditis, reproductive disease syndromes, polyarthritis, mastitis, ocular disease, and septicemia. The only known habitats of H. somni are the mucosal surfaces of ruminants, making this bacterium an opportunistic pathogen. Although it is capable of causing inflammation at systemic sites and is toxic to epithelial and phagocytic cells, the bacterium's wide array of virulence factors act primarily as a defense against, or to escape recognition from, host innate and adaptive immunity.
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