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Inhibition of sexual behavior in nai...

Foster, Nicholas N.

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Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb.

Record Type:	Electronic resources : Monograph/item
Title/Author:	Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb./
Author:	Foster, Nicholas N.
Published:	Ann Arbor : ProQuest Dissertations & Theses, : 2006,
Description:	30 p.
Notes:	Source: Masters Abstracts International, Volume: 68-11.
Contained By:	Masters Abstracts International68-11.
Subject:	Neurology. -
Online resource:	https://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=1441020
ISBN:	9781109798111

Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb.
Foster, Nicholas N.

Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb. - Ann Arbor : ProQuest Dissertations & Theses, 2006 - 30 p.

Source: Masters Abstracts International, Volume: 68-11.

Thesis (M.A.)--University of Southern California, 2006.

Arginine vasopressin (AVP) is a neuropeptide known to influence many rodent social behaviors. Experiment 1 assessed the effects on sexual behavior of AVP (1 ng) infusions into the olfactory bulbs of male rats, finding that AVP significantly inhibited the number of intromissions displayed compared to control animals. Experiment 2 sought to expand upon these findings by comparing groups receiving injections of AVP, a V1b vasopressin receptor antagonist, both AVP and the antagonist (1 ng in all cases), or vehicle. Additionally, immediately after testing, brain tissue samples were collected and were assayed for c-Fos expression in the medial amygdala. AVP alone and paired with the antagonist caused significant reductions in intromission. All drug-treated groups expressed significantly less c-Fos than controls. A model of olfactory processing is proposed whereby the antagonist depresses the function of mitral cells, the primary olfactory bulb efferents, while AVP activates granule cells, which inhibit mitral cells.

ISBN: 9781109798111Subjects--Topical Terms:

588698
Neurology.

Inhibition of sexual behavior in naive male rats by infusion of vasopressin or V1b vasopressin receptor antagonist into the olfactory bulb.
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500 $a Publisher info.: Dissertation/Thesis.
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502 $a Thesis (M.A.)--University of Southern California, 2006.
520 $a Arginine vasopressin (AVP) is a neuropeptide known to influence many rodent social behaviors. Experiment 1 assessed the effects on sexual behavior of AVP (1 ng) infusions into the olfactory bulbs of male rats, finding that AVP significantly inhibited the number of intromissions displayed compared to control animals. Experiment 2 sought to expand upon these findings by comparing groups receiving injections of AVP, a V1b vasopressin receptor antagonist, both AVP and the antagonist (1 ng in all cases), or vehicle. Additionally, immediately after testing, brain tissue samples were collected and were assayed for c-Fos expression in the medial amygdala. AVP alone and paired with the antagonist caused significant reductions in intromission. All drug-treated groups expressed significantly less c-Fos than controls. A model of olfactory processing is proposed whereby the antagonist depresses the function of mitral cells, the primary olfactory bulb efferents, while AVP activates granule cells, which inhibit mitral cells.
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