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Frontal lobe functions and eating pa...
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Groff, April R.
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Frontal lobe functions and eating pathology.
紀錄類型:
書目-電子資源 : Monograph/item
正題名/作者:
Frontal lobe functions and eating pathology./
作者:
Groff, April R.
出版者:
Ann Arbor : ProQuest Dissertations & Theses, : 2007,
面頁冊數:
88 p.
附註:
Source: Dissertations Abstracts International, Volume: 69-08, Section: B.
Contained By:
Dissertations Abstracts International69-08B.
標題:
Psychotherapy. -
電子資源:
https://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3279924
ISBN:
9780549211198
Frontal lobe functions and eating pathology.
Groff, April R.
Frontal lobe functions and eating pathology.
- Ann Arbor : ProQuest Dissertations & Theses, 2007 - 88 p.
Source: Dissertations Abstracts International, Volume: 69-08, Section: B.
Thesis (Ph.D.)--Boston University, 2007.
This item must not be sold to any third party vendors.
Converging evidence suggests an association between frontal lobe functioning and eating pathology. The present study had multiple aims. The first aim was to compare performances of individuals with significant eating pathology to control participants on neuropsychological tasks sensitive to frontal lobe functioning. The second aim of this study was to explore whether individuals with eating pathology reported more symptoms of disorders reflective of deficits in frontal lobe functioning compared to control subjects. The third aim was to determine if neuropsychological factors contribute to the prediction of eating pathology when previously established risk factors are considered. Performances on a neuropsychological test battery were compared between 51 non-treatment-seeking women with significant eating pathology and 51 control participants who had little or no reported eating pathology. Results indicated significant group differences across three of four neuropsychological factors of frontal lobe function: Strategy Implementation, Divided Attention, and Mental Flexibility. A continuous measure of eating pathology was also significantly correlated with Divided Attention, Mental Flexibility, and Behavioral Inhibition factors. The two groups did not differ on general cognitive measures, and scores on these general cognitive measures did not correlate significantly with eating pathology scores. The experimental group endorsed significantly more symptoms of current obsessive-compulsive disorder and current attention deficit hyperactivity disorder (ADHD), as well as childhood ADHD, than control participants. Eating pathology scores were also significantly positively correlated with scores on the three symptom inventory measures. Sequential regression analysis revealed that when established risk factors for eating pathology were accounted for statistically, factors of frontal functioning significantly contributed to the overall variance in eating pathology scores. Thus, when negative affect, exposure to individuals with eating disorders and body dissatisfaction were statistically controlled, Divided Attention and Behavioral Inhibition contributed significantly to the prediction of eating pathology scores. Overall, results suggest that eating pathology is associated with specific disturbances in frontal lobe functioning, that concurrent symptoms consonant with frontal deficits may be present, and inclusion of these deficits may improve models of eating pathology. More prospective, longitudinal studies including specific neurocognitive factors in addition to social and psychological influences in the development of eating pathology are needed.
ISBN: 9780549211198Subjects--Topical Terms:
519158
Psychotherapy.
Subjects--Index Terms:
Attention deficit hyperactivity disorder
Frontal lobe functions and eating pathology.
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Converging evidence suggests an association between frontal lobe functioning and eating pathology. The present study had multiple aims. The first aim was to compare performances of individuals with significant eating pathology to control participants on neuropsychological tasks sensitive to frontal lobe functioning. The second aim of this study was to explore whether individuals with eating pathology reported more symptoms of disorders reflective of deficits in frontal lobe functioning compared to control subjects. The third aim was to determine if neuropsychological factors contribute to the prediction of eating pathology when previously established risk factors are considered. Performances on a neuropsychological test battery were compared between 51 non-treatment-seeking women with significant eating pathology and 51 control participants who had little or no reported eating pathology. Results indicated significant group differences across three of four neuropsychological factors of frontal lobe function: Strategy Implementation, Divided Attention, and Mental Flexibility. A continuous measure of eating pathology was also significantly correlated with Divided Attention, Mental Flexibility, and Behavioral Inhibition factors. The two groups did not differ on general cognitive measures, and scores on these general cognitive measures did not correlate significantly with eating pathology scores. The experimental group endorsed significantly more symptoms of current obsessive-compulsive disorder and current attention deficit hyperactivity disorder (ADHD), as well as childhood ADHD, than control participants. Eating pathology scores were also significantly positively correlated with scores on the three symptom inventory measures. Sequential regression analysis revealed that when established risk factors for eating pathology were accounted for statistically, factors of frontal functioning significantly contributed to the overall variance in eating pathology scores. Thus, when negative affect, exposure to individuals with eating disorders and body dissatisfaction were statistically controlled, Divided Attention and Behavioral Inhibition contributed significantly to the prediction of eating pathology scores. Overall, results suggest that eating pathology is associated with specific disturbances in frontal lobe functioning, that concurrent symptoms consonant with frontal deficits may be present, and inclusion of these deficits may improve models of eating pathology. More prospective, longitudinal studies including specific neurocognitive factors in addition to social and psychological influences in the development of eating pathology are needed.
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